The next stage of our research is to conduct an intervention study, in which a program of exposure controls will be rolled out in several collision repair workshops.
This will involve improvements to extraction and ventilation, improved use of protective gear, and worker training. On the basis of this evidence, a comprehensive exposure control and health protection program tailored to the collision repair industry in New Zealand will be developed, with a view to implementing it industry-wide and potentially across other sectors where workers are at risk of solvent exposure.
Portsmouth Climate Festival — Portsmouth, Portsmouth. Edition: Available editions United Kingdom. Become an author Sign up as a reader Sign in. New research suggests that spray painters and panel beaters could be at higher risk of health effects through solvent exposure. New Zealand stories Brain health.
Kinetics of 1,1,1-trichloroethane in volunteers; influence of exposure concentration and work load. Int Arch Occup Environ Health 42 NIOSH a. Criteria for a recommended standard: occupational exposure to toluene.
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Acta Neurol Scand 42 Exposures at the STEL should not be longer than 15 min and should not be repeated more than four times per day. Carbon disulfide is used as a solvent for a variety of fats, oils, waxes, and resins NIOSH b , and it has been known since the 19th century to cause psychosis and peripheral neuropathy in exposed workers Seppalainen et al. Chronic exposure to CS 2 at workplace concentrations of approximately 20 to 40 ppm produced psychomotor deficits and impaired intellectual function in viscose rayon workers compared with unexposed workers Seppalainen and Haltia ; Hanninen ; Hanninen et al.
Experimental animal studies have confirmed that CS 2 is a neurotoxicant Spencer and Schaumburg Dogs exposed to approximately ppm CS 2 by inhalation for 10 to 15 weeks developed behavioral disturbances, ataxia, and paralysis Lewey et al. Male and female Sprague-Dawley rats and male New Zealand rabbits exposed to ppm CS 2 by inhalation for 22 and 10 weeks, respectively, showed slowed nerve conduction velocities and weakness of hind limbs Seppalainen and Linnoila , They developed pathophysiologic changes in the central and peripheral nervous systems giant axonal swelling and fiber degeneration similar to those seen in humans with n-hexane neuropathy.
Inhalation exposure to 1, ppm methyl ethyl ketone MEK in combination with 9, ppm n-hexane for 15 weeks potentiated the neurotoxicity of n-hexane in male Wistar rats when compared with exposures to 10, ppm n-hexane alone for the same period Altenkirch et al. Methyl n-butyl ketone is used commercially as a solvent for lacquers, nitrocellulose, resins, oils, fats, and waxes; it is also used in varnish and lacquer removers NIOSH a. Continuous MBK inhalation exposure of chickens to ppm and of cats and Sprague-Dawley rats to ppm for 12 weeks produced a pattern of neurologic damage focal axonal swelling and myelin sheath degeneration similar to that seen in workers Mendell et al.
This solvent has caused a generally reversible cranial and peripheral neuropathy associated with sensory loss and motor weakness in the trigeminal nerve and, to a smaller extent, in the facial and optic cranial nerves Feldman et al. Volunteers exposed for two 4-hr periods at average concentrations of ppm showed statistically significant decreases in performance ability on tests of perception, reaction time, memory, and manual dexterity compared with their performance ability before exposure Salvini et al.
Chronic occupational exposure at concentrations above ppm Politis et al. No adequate epidemiologic studies or experimental animal studies have yet been conducted to corroborate these reports Spencer and Schaumburg In male and female Mongolian gerbils, 3 months of inhalation exposure to 60 ppm trichloroethylene followed by a 4-month recovery period produced increases in brain protein levels associated with brain damage Haglid et al.
In male and female Mongolian gerbils, 3 months of inhalation exposure to 60 ppm perchloroethylene followed by a 4-month recovery period produced increases in brain protein levels associated with brain damage Haglid et al. Toluene abuse e. This damage begins with Type 1 neurobehavioral changes and progresses to Type 3 chronic toxic encephalopathy see Table 1 of text , including impairment of intellect and memory and evidence of CNS atrophy Lazar et al.
High levels of solvent vapours can quickly build up in poorly ventilated spaces, especially in warmer temperatures. Effects of acute inhalation poisoning may include:. Once a person is moved to clear, fresh air, the effects of inhalation poisoning will generally rapidly disappear. If problems persist, seek medical advice. Always seek medical help for someone who has lost consciousness from poisoning, even if they seem to recover. Spraying puts all the solvents in coatings into the air quickly.
Most solvents have similar health effects They can harm many parts of your body including your brain, skin, liver, kidneys and respiratory system. This is what solvents can do to your skin. What if you get solvent on your skin? Read more about gloves Your browser is antiquated and no longer supported on this website.
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